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A similar pathway was shown to be involved in the release of insulin in response to TGR5 ligands in pancreatic cells (Kumar et al., Figure 5 Inhibition of OA-induced GLP-1 and PYY release by H 2 S To investigate the regulation of TGR5-mediated GLP-1 and PYY release by H 2 S, cells were incubated with L-cysteine, an activator of endogenous enzymes CSE and CBS, for 10 min and then GLP-1 and PYY release in response to OA was measured

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433 Rather than producing from mitochondrial TCA cycle under controlled conditions, the NADH in diabetic islets is generated by cytoplasmic sorbitol oxidation and mitochondrial pyruvate oxidation in response to diabetes and hyperglycemia stimulate

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