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The findings revealed that irregular activation of HGF/c-MET enhanced TGF- release from cancer cells, reduced CTL penetration at the tumor site, and eventually caused immune evasion

59 , 60 , 61 , 62 The reversible oxidative modifications of cysteine residues imply an important mechanism whereby ROS and RNS can readily interact with these molecules, dictating protein function and cell signaling that can be regulated by GSH

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